[HTML][HTML] Treg cells mediate recovery from EAE by controlling effector T cell proliferation and motility in the CNS

M Koutrolos, K Berer, N Kawakami, H Wekerle… - Acta neuropathologica …, 2014 - Springer
M Koutrolos, K Berer, N Kawakami, H Wekerle, G Krishnamoorthy
Acta neuropathologica communications, 2014Springer
Regulatory T cells are crucial in controlling various functions of effector T cells during
experimental autoimmune encephalomyelitis. While regulatory T cells are reported to exert
their immunomodulatory effects in the peripheral immune organs, their role within the central
nervous system (CNS) during experimental autoimmune encephalomyelitis is unclear. Here,
by combining a selectively timed regulatory T cells depletion with 2-photon microscopy, we
report that regulatory T cells exercise their dynamic control over effector T cells in the CNS …
Abstract
Regulatory T cells are crucial in controlling various functions of effector T cells during experimental autoimmune encephalomyelitis. While regulatory T cells are reported to exert their immunomodulatory effects in the peripheral immune organs, their role within the central nervous system (CNS) during experimental autoimmune encephalomyelitis is unclear. Here, by combining a selectively timed regulatory T cells depletion with 2-photon microscopy, we report that regulatory T cells exercise their dynamic control over effector T cells in the CNS. Acute depletion of regulatory T cells exacerbated experimental autoimmune encephalomyelitis severity which was accompanied by increased pro-inflammatory cytokine production and proliferation of effector T cells. Intravital microscopy revealed that, in the absence of regulatory T cells, the velocity of effector T cells was decreased with simultaneous increase in the proportion of stationary phase cells in the CNS. Based on these data, we conclude that regulatory T cells mediate recovery from experimental autoimmune encephalomyelitis by controlling cytokine production, proliferation and motility of effector T cells in the CNS.
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